A transient receptor potential-like channel mediates synaptic transmission in rod bipolar cells.GPR179 is required for depolarizing bipolar cell function and is mutated in autosomal-recessive complete congenital stationary night blindness.Ultrastructural localization of GPR179, and the impact of mutant forms on retinal function in CSNB1 patients and a mouse model.Mutations in TRPM1 are a common cause of complete congenital stationary night blindness.
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R.G. Gregg